I didn’t intend to translate Prof Kojima’s articles three posts in a row, but since Ashmedai already wrote about the study by a Japanese cardiologist whose patients’ mortality rate doubled after getting the Covid jab, I guess I might as well do so. Prof Kojima’s article is titled “
Some other friends of mine in Tokyo are working on vial content analysis in conjunction with batch analysis in the hopes of verifying the 'whys and hows' batch/lot adverse events appeared to vary significantly from lot to lot. Very clever bunch of researchers indeed.
I am all for keeping an open mind, but any investigation which attempts to assign causation of adverse events to "bad batches" raises doubts in my mind. As Bret Weinstein has noted, a simple explanation of the mechanism of injury is that the mRNA technology forces the body's own cells to produce a particle (here the spike protein) that causes an autoimmune response. The body's T-cells kill our own cells that present the foreign particle. The minute it was established that the mRNA does not remain locally in the injection site, there was a clear plausible explanation for injuries. After all it's one thing to kill off some muscle cells in one's shoulder in order to develop an effective immune response, but it is another thing to trigger the body's immune system to attack various organs and systems in an "autoimmune cascade."
Paul Offit has attempted to "save" the mRNA platform by suggesting that the problem was develoeprs selected the wrong particle--the spike protein. So he blames the injection, but asserts the next time mRNA developers will get it right. But if the entire platform is the cause of the injury because of the obvious autoimmune challenges it creates, then mRNA "vaccination" is likely to fail every time.
The "bad batch" argument is similar. It solely places blame on the manufacturing process and acquits the mRNA platform. To be sure there are other plausible ideas about mechanism of injury such as Mark Girardot's "bolus" hypothesis--where failure to aspirate the needle to assure that a blood vessel has not been intersected results in a large "bolus" of product directly reaching the heart and other areas of the body. But the autoimmune hypothesis is strong, and so though I am open to "hot lots," I remain skeptical.
Cheers Guy, great work, and thanks as always!
Some other friends of mine in Tokyo are working on vial content analysis in conjunction with batch analysis in the hopes of verifying the 'whys and hows' batch/lot adverse events appeared to vary significantly from lot to lot. Very clever bunch of researchers indeed.
And there is always this site: https://howbad.info/index.html
Take care fella and have a great week.
I am all for keeping an open mind, but any investigation which attempts to assign causation of adverse events to "bad batches" raises doubts in my mind. As Bret Weinstein has noted, a simple explanation of the mechanism of injury is that the mRNA technology forces the body's own cells to produce a particle (here the spike protein) that causes an autoimmune response. The body's T-cells kill our own cells that present the foreign particle. The minute it was established that the mRNA does not remain locally in the injection site, there was a clear plausible explanation for injuries. After all it's one thing to kill off some muscle cells in one's shoulder in order to develop an effective immune response, but it is another thing to trigger the body's immune system to attack various organs and systems in an "autoimmune cascade."
Paul Offit has attempted to "save" the mRNA platform by suggesting that the problem was develoeprs selected the wrong particle--the spike protein. So he blames the injection, but asserts the next time mRNA developers will get it right. But if the entire platform is the cause of the injury because of the obvious autoimmune challenges it creates, then mRNA "vaccination" is likely to fail every time.
The "bad batch" argument is similar. It solely places blame on the manufacturing process and acquits the mRNA platform. To be sure there are other plausible ideas about mechanism of injury such as Mark Girardot's "bolus" hypothesis--where failure to aspirate the needle to assure that a blood vessel has not been intersected results in a large "bolus" of product directly reaching the heart and other areas of the body. But the autoimmune hypothesis is strong, and so though I am open to "hot lots," I remain skeptical.
The data should be stratified by adverse event type (infections or other).
Most "bad batches" were deployed during COVID waves. Batches that were distributed early on also went towards elders and disabled people.
Once the data are stratified by adverse event type and age adjusted, very little remains in terms of batch differences.
Excellent work, thank you.
And they are more welcome than ever;
https://www.japantimes.co.jp/news/2023/11/14/japan/science-health/japan-new-drug-trials/
vial content = vile content Pfizer= miser Lethal injection via Russian roulette? No thanks!